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LLLT and Hypothyroidism

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Wed, 03/08/2017 - 10:14 -- Kalon Prensky

Can Low Level Lasers be used to support the Thyroid?

This is a common question, which has always been difficult to answer. Namely because some sources indicate that the Thyroid may be light sensitive, however I have found no concrete evidence that points to actual negative effects of low level laser therapy around the Thyroid. To date there is still no such thing as a low level laser that is FDA cleared for Thyroid treatment. However, that may change some day.
 
* The following information comes from a Study in Brazil, which shows quite promising results:
 
 
Lasers Med Sci. 2012 Jun 21. [Epub ahead of print]
 
Low-level laser in the treatment of patients with hypothyroidism induced by chronic autoimmune thyroiditis: a randomized, placebo-controlled clinical trial.
 
Höfling DB, Chavantes MC, Juliano AG, Cerri GG, Knobel M, Yoshimura EM, Chammas MC.
 
Source
 
Ultrasound Unit, Department of Radiology, University of Sao Paulo Medical School, Hospital das Clínicas, São Paulo, 05403-000, Brazil, dbhofling@uol.com.br.
 
Abstract
 
Chronic autoimmune thyroiditis (CAT) is the most common cause of acquired hypothyroidism, which requires lifelong levothyroxine replacement therapy.
 
Currently, no effective therapy is available for CAT. Thus, the objective of this study was to evaluate the efficacy of low-level laser therapy (LLLT) in patients with CAT-induced hypothyroidism by testing thyroid function, thyroid peroxidase antibodies (TPOAb), thyroglobulin antibodies (TgAb), and ultrasonographic echogenicity.
 
A randomized, placebo-controlled trial with a 9-month follow-up was conducted from 2006 to 2009. Forty-three patients with a history of levothyroxine therapy for CAT-induced hypothyroidism were randomly assigned to receive either 10 sessions of LLLT (830 nm, output power of 50 mW, and fluence of 707 J/cm(2); L group, n = 23) or 10 sessions of a placebo treatment (P group, n = 20).
 
The levothyroxine was suspended 30 days after the LLLT or placebo procedures. Thyroid function was estimated by the levothyroxine dose required to achieve normal concentrations of T(3), T(4), free-T(4) (fT(4)), and thyrotropin after 9 months of postlevothyroxine withdrawal. Autoimmunity was assessed by measuring the TPOAb and TgAb levels.
 
A quantitative computerized echogenicity analysis was performed pre- and 30 days postintervention. The results showed a significant difference in the mean levothyroxine dose required to treat the hypothyroidism between the L group (38.59 ± 20.22 μg/day) and the P group (106.88 ± 22.90 μg/day, P < 0.001). Lower TPOAb (P = 0.043) and greater echogenicity (P < 0.001) were also noted in the L group. No TgAb difference was observed.
 
These findings suggest that LLLT was effective at improving thyroid function, promoting reduced TPOAb-mediated autoimmunity and increasing thyroid echogenicity in patients with CAT hypothyroidism.
 
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*ANOTHER HYPOTHYROID STUDY

Photomed Laser Surg.  2014 Aug;32(8):444-9. doi: 10.1089/pho.2014.3716.

Effects of low-level laser therapy on the serum TGF-β1 concentrations in individuals with autoimmune thyroiditis.

Höfling DB , Chavantes MC, Acencio MM, Cerri GG, Marui S, Yoshimura EM, Chammas MC.

Abstract

OBJECTIVE: The objective of this study was to evaluate the serum concentration of transforming growth factor-β1 (TGF-β1) after low-level laser therapy (LLLT) in patients with hypothyroidism resulting from chronic autoimmune thyroiditis (CAT).

BACKGROUND DATA: Certain data indicate that LLLT is effective in patients with hypothyroidism caused by CAT; however, the mechanisms of action of LLLT in thyroid tissue are unknown. Cytokines could play a role in the response to LLLT.

METHODS: A randomized, placebo-controlled trial included 43 patients with a history of levothyroxine therapy for CAT-induced hypothyroidism. The patients were randomly assigned to receive either 10 sessions of LLLT (830 nm, 50 mW output power, and 707 J/cm(2) fluence; L group, n=23) or 10 sessions of a placebo treatment (P group, n=20) twice a week. Levothyroxine was maintained at the same dose during the entire study period. TGF-β1 was measured both pre-intervention and 30 days post-intervention in both groups. The differences were calculated between the TGF-β1 values observed 30 days post-intervention and the pre-intervention TGF-β1 values for each group (intragroup).

RESULTS: Comparing the differences in TGF-β1 levels between the L group (874.9±541.7 pg/mL) and the P group (-128.4±832.8 pg/mL) revealed that there was a statistically significant increase in TGF-β1 levels 30 days post-intervention in group L compared with the placebo group (p=0.0379).

CONCLUSIONS: This finding suggested that the significant increase in serum TGF-β1 levels in patients with CAT-induced hypothyroidism was associated with the thyroid LLLT procedure. Future studies of the effect of LLLT on TGF-β1 gene expression in thyroid tissue are necessary to confirm these findings.

SOURCE: ClinicalTrials.gov NCT01129492.

 

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*ANOTHER THYROID STUDY

International Scholarly Research Network  ISRN Endocrinology

Volume 2012, Article ID 126720, 9 pages - Resulted in the following Conclusion:

 

In conclusion, the study of the thyroid gland using

CDU showed that LLLT improved gland vascularization and

increased the systolic peak velocity of the inferior thyroid

arteries in patients with CAT-induced hypothyroidism.

 

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